← OncoDesk·MedDesk·web edition — reference only, not medical advice
all sections · full handbook

6Testicular / Germ Cell

Snapshot. The most curable solid tumour in adults — >95% overall, and still curable AT RELAPSE. That single fact drives everything: never dose-reduce, never delay salvage, never treat a relapse as palliative. Two lineages: SEMINOMA vs NSGCT (non-seminomatous germ-cell tumour). Markers: AFP (alpha-fetoprotein) — NEVER raised in pure seminoma, β-hCG (beta-human chorionic gonadotropin), LDH (lactate dehydrogenase). ⚠ A raised AFP in a "seminoma" means it is NOT a seminoma — treat as NSGCT. Metastatic disease is risk-stratified by IGCCCG (International Germ Cell Cancer Collaborative Group) risk group — good / intermediate / poor.
Workup
Scrotal ultrasound + markers (AFP, β-hCG, LDH) BEFORE surgery · RADICAL INGUINAL ORCHIDECTOMY — one act, two jobs: it is the diagnosis (histology, T-stage, LVI [lymphovascular invasion]) and the treatment of the primary; never trans-scrotal biopsy — seeds the nodal basin · markers REPEATED after surgery, allowing for half-life decay (AFP ~5–7 days, β-hCG ~1–3 days) — failure to normalise = occult systemic disease, whatever the scan shows · CT chest/abdomen/pelvis · brain MRI if poor-risk, very high β-hCG, or extensive lung mets · sperm banking BEFORE any chemotherapy or RPLND (retroperitoneal lymph-node dissection) — time-critical, cannot be done retrospectively.
Treatment by setting
SettingTreatment
STAGE I SEMINOMA
seminoma · markers normalised · CT clear
SURVEILLANCE is preferred cure approaches 99% either way, and surveillance spares the majority who are already cured from any treatment at all · relapse-risk factors: tumour >4 cm, rete testis invasion
If not feasible → single-agent carboplatin AUC 7 ×1 (MRC TE19)
Para-aortic EBRT 20 Gy — now LEAST favoured external-beam radiotherapy — second-malignancy + cardiovascular late effects in men who will live decades
STAGE I NSGCT — no LVI
SURVEILLANCE relapse ~15%, and relapse is still curable
STAGE I NSGCT — LVI PRESENT
LVI on the orchidectomy specimen; relapse risk ~50%
Surveillance remains the preferred option in a fully-informed patient able to adhere to follow-up — relapse, if it happens, is still curable with full-course chemo
Alternative → adjuvant BEP ×1 bleomycin + etoposide + cisplatin — cuts relapse to ~2–3%
Or nerve-sparing RPLND
METASTATIC — GOOD risk
NSGCT: testis/retroperitoneal primary, no non-pulmonary visceral mets, AFP <1000, β-hCG <5000, LDH <1.5× ULN (upper limit of normal) · Seminoma: any primary, normal AFP, no non-pulmonary visceral mets
BEP ×3
Or EP ×4 when bleomycin must be avoided etoposide + cisplatin — pre-existing lung disease, older age, impaired renal function, or a planned extensive thoracic resection
METASTATIC — INTERMEDIATE / POOR risk
Intermediate: non-pulm. visceral mets (seminoma) or AFP 1000–10 000 / β-hCG 5000–50 000 / LDH 1.5–10× ULN (NSGCT) · Poor (NSGCT only): mediastinal primary, non-pulm. visceral mets, AFP >10 000, β-hCG >50 000, or LDH >10× ULN — there is no poor-risk seminoma
BEP ×4
Bleomycin contraindicated → VIP ×4 etoposide + ifosfamide + cisplatin
⚠ FULMINANT HIGH-VOLUME CHORIOCARCINOMA IS AN EMERGENCY massive β-hCG, pulmonary haemorrhage, respiratory failure → start chemotherapy urgently; consider a reduced-intensity first cycle to avoid tumour-lysis / haemorrhage catastrophe, then escalate to full dose
RESIDUAL MASS after chemotherapy
markers normalised, mass persists
NSGCT → RESECT any residual mass ≥1 cm post-chemo RPLND ± thoracic resection · findings: necrosis/fibrosis, teratoma (chemo-resistant, grows, can transform — must come out), or viable germ-cell tumour (→ consider further chemo)
SEMINOMA → do NOT reflexively operate masses regress slowly
Seminoma, mass >3 cm → FDG-PET at ≥6 weeks negative → observe; positive → biopsy / resect · PET is unreliable in NSGCT — it cannot exclude teratoma
🔴 RELAPSED / REFRACTORY
still curative intent
🔴 The single most important row in the section — relapse here is NOT palliative
Conventional-dose salvage → TIP paclitaxel + ifosfamide + cisplatin — ~65% long-term remission in favourable-risk relapse · VeIP (vinblastine + ifosfamide + cisplatin) ~25%; or VIP
High-dose chemo + autologous stem-cell rescue carboplatin/etoposide, Indiana regimen — as initial salvage in poor-risk relapse, or after failed conventional salvage · refer to a high-volume germ-cell centre
⚠ Late relapse (>2 yr) is chemo-resistant → SURGERY is the mainstay
GROWING TERATOMA SYNDROME
mass enlarging WHILE markers normal/falling, on chemo
Complete surgical resection not progression — chemotherapy will not touch it
BRAIN METASTASES
Platinum-based chemotherapy (BEP-type) PLUS radiotherapy and/or surgery still curative intent, unlike most solid tumours
Surveillance
Markers (AFP, β-hCG, LDH) + imaging on a defined schedule — intensive for the first 2 years (when most relapses occur), tapering to year 5 and beyond. Only a valid strategy if the patient will actually attend — it is the follow-up, not the absence of treatment, that delivers the cure. Also track the contralateral testis (GCNIS [germ-cell neoplasia in situ] / second primary) and long-term cardiovascular, renal, and metabolic late effects.
Watch
SPERM BANKING BEFORE ANY CHEMOTHERAPY OR RPLND — non-negotiable and time-critical. Bleomycin → pulmonary fibrosis (cumulative dose; lifelong caution with high inspired oxygen — flag for any future anaesthetist); flagellate hyperpigmentation (whip-like linear streaks on trunk/neck/back ± pruritus — a classic, benign, dose-related skin marker; document, does NOT mandate stopping bleomycin — distinct from the lung toxicity). Cisplatin → nephrotoxicity, oto- + vestibular toxicity (hearing loss/tinnitus/dizziness — screen before blaming positional vertigo), peripheral neuropathy, hypomagnesaemia (renal Mg wasting), long-term cardiovascular/metabolic risk. Ifosfamide → haemorrhagic cystitis (give MESNA) and encephalopathy. RPLND → retrograde ejaculation (nerve-sparing reduces it).
⚠ GERM-CELL CHEMOTHERAPY IS CURATIVE — PROTECT THE DOSE INTENSITY: low counts on a cycle day → DELAY to ANC (absolute neutrophil count) recovery and give the FULL dose, add G-CSF secondary prophylaxis — NEVER dose-reduce BEP. Track markers every cycle — decline is the efficacy readout; markers rising on treatment means resistance, not a blip.
← ColorectalProstate →